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The downstream effects are distinct from incretin signaling: slowing of gastric emptying, suppression of postprandial glucagon, and reduction of food intake through central satiety circuits that do not heavily overlap with GLP-1 pathways

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While timing of puberty is genetically controlled (11), it is likely that the development of the body to a specific weight and/or size through the anabolic actions of GH and IGFs is at least partly responsible for onset of puberty (10, 12)